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The first serious attempt to find signals from another civilization was initiated by Dr. Frank Drake using a radio telescope at Green Bank, West Virginia, in 1959. Drake and his colleagues called the undertaking Project Ozma, named after the fantasy land of Oz. The scientific establishment regarded Project Ozma with interest, amusement, and some skepticism. But Drake believed that if enough stars were scanned with the sensitive radio receivers and large antennas at Green Bank and other radio observatories, it was only a matter of time before signals from an extraterrestrial civilization were picked up. Some of the stars that Drake investigated were Tau Ceti in the constellation Cetus and Epsilon Eridani in the constellation Eridanus. Various frequencies were checked, but especially those in the vicinity of the use .net framework crystal report bar code writer to develop barcode in .net colored BusinessRefinery.com/barcodeuse word documents bar code drawer to assign barcodes in word documents barcodes BusinessRefinery.com/barcodeSupportive; ventilation 3,4-DAP Possibly IVIG
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The ophthalmoplegia, whether occurring alone or with weakness or ataxia of other parts, is almost uniformly associated with a speci c antineural antibody, anti-GQ1b. The ophthalmoplegic pattern raises the possibility of myasthenia gravis, botulism, diphtheria, tick paralysis, and basilar artery occlusion. Bilateral but asymmetrical facial and abducens weakness, coupled with distal paresthesias or with proximal leg weakness, are other fairly common variants in our experience (Ropper, 1994). The tendon re exes may be absent initially only at the ankles or at the knees. Lyme disease and sarcoidosis are then considerations in diagnosis. Paraparetic, ataxic, and purely motor or purely sensory forms of the illness have also been observed. Less dif culty attends these diagnoses if paresthesias in the acral extremities, progressive reduction or loss of re exes, and relative symmetry of weakness appear after several days of signs. The laboratory tests that af rm the diagnosis of typical GBS give similar but generally milder abnormalities if they are carefully sought in all these variant forms. There has been a recent tendency to separate a group of cases with presumed diffuse axonal damage on the basis of an abrupt and explosive onset, severe paralysis, minor sensory features, and the electrophysiologic nding of inexcitability of nerves. This axonal type of GBS represents 5 percent (or fewer) of cases but its identi cation has some value in that a poor response to treatment and very protracted recovery are to be expected in most cases. This variant is described later. In a few patients the weakness continues to evolve for 3 to 4 weeks or even longer. From this group a chronic form of demyelinative neuropathy (chronic in ammatory demyelinating polyneuropathy, or CIDP) often emerges and an intermediate group that progress for 4 to 8 weeks and then improve can be identi ed (see further on). Acute Axonal Form of Guillain-Barre Syndrome This subject has been alluded to earlier. Feasby and colleagues drew attention to an acute are exic polyneuropathy clinically similar to GBS but characterized pathologically by widespread and severe axonal degeneration. In their initial report they described ve patients, all with a rapid evolution of polyneuropathy and very slow and poor recovery. Unlike the common form of GBS, muscle atrophy in these patients became apparent relatively early in the disease (within weeks). 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