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BAROHN RJ, JACKSON CE, ROGERS SJ, et al: Prolonged paralysis due to non-depolarizing neuromuscular blocking agents and corticosteroids. Muscle Nerve 17:647, 1994. BRAAKHEKKE JP, DE BRUIN MI, STEGEMAN DF, et al: The second wind phenomenon in McArdle s disease. Brain 109:1087, 1986. CONN JW: Aldosteronism in man: Some clinical and climatological aspects. JAMA 183:871, 1963. CORI GT, CORI CF: Glucose-6-phosphatase of the liver in glycogen storage disease. J Biol Chem 199:661, 1952. CURRY SC, CHANG D, CONNOR D: Drug and toxin-induced rhabdomyolysis. Ann Emerg Med 18:1068, 1989. DIDONATO S, TARONI F: Disorders of lipid metabolism, in Engel AG, Franzini-Armstrong C (eds): Myology, 3rd ed. New York, McGraw-Hill, 2004, pp 1587 1622. DIMAURO S, MELIS-DIMAURO P: Muscle carnitine palmitoyltransferase de ciency and myoglobinuria. Science 182:929, 1973. DIMAURO S, TONIN P, SERVIDEI S: Metabolic myopathies, in Vinken PJ, Bruyn GW (eds): Handbook of Clinical Neurology, vol 18. Amsterdam, North Holland, 1992, pp 479 526. DRESNER SC, KENNERDELL JS: Dysthyroid orbitopathy. Neurology 35: 1628, 1985. DUBOIS DC, ALMON RR: A possible role for glucocorticoids in denervation atrophy. Muscle Nerve 4:370, 1981. ENGEL AG, ANGELINI C: Carnitine de ciency of human skeletal muscle with associated lipid storage myopathy: A new syndrome. Science 179:899, 1973. ENGEL AG, FRANZINI-ARMSTRONG C (eds): Myology, 3rd ed. New York, McGraw-Hill, 2004. ENGEL WK, VICK NK, GLUECK J, LEVY RI: A skeletal muscle disorder associated with intermittent symptoms and a possible defect in lipid metabolism. N Engl J Med 282:697, 1970. FARIS AA, REYES MG: Reappraisal of alcoholic myopathy: Clinical and biopsy study on chronic alcoholics without muscle weakness or wasting. J Neurol Neurosurg Psychiatry 34:86, 1971. FARMER JA: Learning from the cerivastatin experience. Lancet 358:1383, 2001. GORSON KC, ROPPER AH: Generalized paralysis in the intensive care unit: Emphasis on the complications of neuromuscular blocking agents and corticosteroids. J Int Care Med 11:219, 1996. GRIGGS RC, MENDELL JR, MILLER RG: Evaluation and Treatment of Myopathies. Philadelphia, Davis, 1995. HALLER RG, DRACHMAN DB: Alcoholic rhabdomyolysis: An experimental model in the rat. Science 208:412, 1980.
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Figure 2-5. Short-latency brainstem auditory evoked responses (BAERs). Diagram of the proposed electrophysiologic-anatomic correlations in human subjects. Waves I through V are the ones measured in clinical practice.
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7. What is the intersection of these two sets What is their union A = {1, 1/2, 1/3, 1/4, 1/5, 1/6, ...} G = {1, 1/2, 1/4, 1/8, 1/16, 1/32, ...} In set A, the denominator of the fraction increases by 1 as you go down the list. In set G, the denominator doubles as you go down the list. All the numerators in both sets are equal to 1. 8. List all the subsets of {1, 2, 3}. Here s a hint: Whenever you want to find all the subsets of a small set like this, first list its individual elements. Then make up every possible set that contains at least one of those elements. Finally, be sure to include the empty set, which is a subset of any other set. 9. List all the subsets of {1, {2, 3}}. Be careful. The hint given with Problem 8 is important here. 10. List all the subsets of {1, {2, {3}}}. Be extra careful! The hint given with Problem 8 is even more important here.
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So first, if I look at this dashboard, the one area that I would really like to point out is the fact that the color scheme doesn t match. If you look at the screen on the far right you ll notice that it s identifying each of the sales organizations as a specific color. Now, if I look at any of the charts I don t see this color scheme anywhere. So that s our first issue and really the one that I wanted to highlight in this dashboard, but if we continue to review these screens we see a few more concerns. Second, if I look at the different charts I can t really tell which ones are the primary KPIs. They are all in the same font and colors and there s nothing distinguishing one metric from the other in importance. Third, in the upper-middle chart I m wondering what is involved in the organizations of each of these Sales Orgs. I don t see that they are sorted alphabetically, or in priority of total sales, or by technical name. Basically they are listed randomly and the business analyst will have an issue with that setup. Finally, I would also take note of the titles titling something Series 1 is not a title. So I ve assumed that this is a graphical interpretation of Revenue YTD from the chart on the left but that s just an assumption and to do that assumption your business user will have to think about something else other than analysis of the data. Again, remember our jobs here are to collect the data and present the data consistently so that the business analysts will do their jobs of analysis of the data. I will also assume that the lower chart has the primary KPIs since it takes up most of the room on the dashboard and we should focus on that one the most. Therefore, having to be able to identify the KPI itself quickly is critical. In this case the title again is not straightforward enough and needs to be cleaned up. I would also think that we need to have some sort of context to this information. Are the sales for this time period good, okay, bad For that purpose we need to have some sort of benchmark Planned Sales, Period to Period comparison, or something like that to give the information more validity.
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The phenomenon of myoclonus has already been discussed in Chap. 6, where the relationship to seizures was indicated. Characterized by a brusque, brief, muscular contraction, some myoclonic jerks are so small as to involve only one muscle or part of a muscle; others are so large as to implicate a limb on one or both sides of the body or the entire trunk musculature. Many are brief, lasting 50 to 100 ms. They may occur intermittently and unpredictably or present as a single jerk or a brief salvo. As mentioned earlier, an outbreak of several small, rhythmic myoclonic jerks may appear with varying frequency as part of absence seizures and as isolated events in patients with generalized clonic-tonic-clonic or tonic-clonic seizures. As a rule, these types of myoclonus are quite benign and respond well to medication. In contrast, disseminated myoclonus (polymyoclonus), having its onset in childhood, raises the suspicion of acute viral encephalitis, the myoclonus-opsoclonus-ataxia syndrome of Kinsbourne, lithium or other drug toxicity, or, if lasting a few weeks, subacute sclerosing panencephalitis. Chronic progressive polymyoclonus with dementia characterizes the group of juvenile lipidosis, Lafora-
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