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Therefore, by comparison with eqn. (4.49), a suitable linear system gyro error model is 20 (4.53) G(s) = 10 + s and the PSD of the corresponding white driving noise process wg is Swg (j ) = 0.012 rad/s2 Hz
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Following a method similar to that of Section 10.5.2.1, to derive the dynamic model for P, set the derivative of eqn. (10.32) equal to the left side of eqn. (10.46): PRn + (I + P)Rn b b which simpli es as follows, PRn + (I + P)Rn b b = (I + P)Rn ( b b + b ) in ib b ib n b n = (I + P)Rb + (I + P)Rb ib Rn b b in b b n n b b P = Rb ib Rn Rb in Rn (10.54) = (I + P)Rn ( b b ) ib in b (10.53)
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Figure 4.22 Tumescing the face the lower face (Figure 4.22). Using smaller gauge cannulas (sizes 14 and 16), neck liposuction is performed from stab incisions performed in the submentum and the infraauricular regions. The liposuction is carefully performed above the mandible to create a more optimal contour. After contouring with liposuction, the incision is then performed. Prior to raising the ap, tunnels are made with the liposuction cannula with the suction switched off. Using either a baby Metzenbaum or Mayo scissors, the ap is undermined to approximately 6 cm anteriorly. The neck is undermined to midline, hermostasis achieved. Plication with 3-0 absorbable or nonabsorbable of the SMAS is then performed (Figure 4.23). The vector of Following informed consent and preoperative photos, the patient is marked. The vectors should be as vertical as possible. Depending on the site, the apex/entry point of the thread should be within the hairline. For neck thread lifts, the entry point is infra-auricular. Tumescent anesthesia is in ltrated. Using a 2.5-mm punch, a small incision is made for the entry point. This area is undermined with tenotomy scissors so that a small pocket is formed. Using a cannula, the marked pathway of the suture as well as adjacent areas are bluntly undermined. This procedure essentially creates a small sliding ap. A CT400 barbed thread on a keith needle is inserted through the entry point, and then guided in the subcutaneous layer in a sinusoidal fashion following the markings. The needle exits just superior to the eyebrows for the browlift, just lateral to the nasolabial folds for the midface lift, and just lateral to midline for the neck lift. The thread should be pulled through until there are no barbs at the entry point. The thread is pulled gently retracted locking the barbs. The suture is cut leaving a whisker. Then the other end of the CT400 is inserted through the same entry point, and the procedure is repeated. Before inserting this needle, a Gore-Tex pledget may be placed onto the thread and positioned on the smooth portion, which will act as an anchor. Depending on the site, several sets of the sutures may need to be placed. The patient is placed in a sitting position, and contouring begins by holding the whiskers and manually pushing up on the lax skin in a superior direction. This is continued until there is a visible lift and bunching of skin at the entry point. The excess sutures are then cut ush with the skin at the exit points. Some practitioners do not cut the suture, but have the patient return on the second and
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ADAMS RD, FOLEY JM: The neurological disorder associated with liver disease. Res Publ Assoc Res Nerv Ment Dis 32:198, 1953. ADAMS RD, VICTOR M, MANCALL EL: Central pontine myelinolysis. Arch Neurol Psychiatry 81:154, 1959. AIKAWA N, SHINOZAWA Y, ISHIBIKI K, et al: Clinical analysis of multiple organ failure in burned patients. Burns 13:103, 1987. ALFREY AC, LEGENDRE GR, KAEHNY WD: The dialysis encephalopathy syndrome: Possible aluminum intoxication. N Engl J Med 294:184, 1976. AMES A, WRIGHT RL, KOWADA M, et al: Cerebral ischemia: II. The nore ow phenomenon. Am J Pathol 52:437, 1968. ARIEFF AI: Hyponatremia associated with permanent brain damage. Adv Intern Med 21:325, 1987. ARIEFF AI: Hyponatremia, convulsions, respiratory arrest, and permanent brain damage after elective surgery in healthy women. N Engl J Med 314:1529, 1986. AUER RN: Progress review: Hypoglycemic brain damage. Stroke 17:699, 1986. AUSTEN FK, CARMICHAEL MW, ADAMS RD: Neurologic manifestations of chronic pulmonary insuf ciency. N Engl J Med 257:579, 1957. BASILE AS, HUGHES RD, HARRISON PM, et al: Elevated brain concentrations of 1,4-benzodiazepines in fulminant hepatic failure. N Engl J Med 325:473, 1991. BERNARD Sa, GRAY TW, BUIST MD, et al: Treatment of comatose survivors of out-of-hospital cardiac arrest with induced hypothermia. N Engl J Med 346:557, 2002. BHATIA MP, BROWN P, GREGORY R, et al: Progressive myoclonic ataxia associated with coeliac disease. Brain 118:1087, 1995. BOLTON CF, YOUNG GB: Neurological Complications of Renal Disease. Boston, Butterworth, 1990. BOLTON C, YOUNG GB, ZOCHODNE DW: The neurological complications of sepsis. Ann Neurol 33:94, 1993. BRAIN L, JELLINEK EH, BALL K: Hashimoto s disease and encephalopathy. Lancet 2:512, 1966. BURCAR PJ, NORENBERG MD, YARNELL PR: Hyponatremia and central pontine myelinosis. Neurology 27:223, 1977. BURN DJ, BATES D: Neurology and the kidney. J Neurol Neurosurg Psychiatry 65:810, 1998. BUTTERWORTH RF, GIGUIERE JF, MICHAUD J, et al: Ammonia: Key factor in the pathogenesis of hepatic encephalopathy. Neurochem Pathol 6:1, 1987. CAO X-Y, JIAN GX-M, DOU Z-H, et al: Timing of vulnerability of the brain to iodine de ciency in endemic cretinism. N Engl J Med 331:1739, 1994. CAVANAGH JB: Liver bypass and the glia, in Plum F (ed): Brain dysfunction in metabolic disorders. Res Publ Assoc Res Nerv Ment Dis 53:13, 1974. CHOI DW, ROTHMAN SM: The role of glutamate neurotoxicity in hypoxicischemic neuronal death. Annu Rev Neurosci 13:171, 1990. CHOI IS: Delayed neurologic sequelae in carbon monoxide intoxication. Arch Neurol 40:433, 1983. CHONG JY, ROWLAND LP, UTIGER RD: Hashimoto encephalopathy: Syndrome or myth Arch Neurol 60:164, 2003. COOKE WT, SMITH WT: Neurologic disorders associated with adult coeliac disease. Brain 89:683, 1966. CREMER GM, GOLDSTINE NP, PARIS J: Myxedema and ataxia. Neurology 19:37, 1969. CROSS AH, GOLUMBEK PT: Neurologic manifestations of celiac disease. Neurology 60:1566, 2003. DAHLQUIST NR, PERRAULT J, CALLAWAY CW: D-Lactic acidosis and encephalopathy after jejunoileostomy: Response to overfeeding and to fasting in humans. Mayo Clin Proc 59:141, 1984. DELONG GR, STANBURY JB, FIERRO-BENITEZ R: Neurological signs in congenital iodine-de ciency disorder (endemic cretinism). Dev Med Child Neurol 27:317, 1985. DIMAURO S, TONIN P, SERVIDEI S: Metabolic myopathies, in Rowland LP, DiMauro S (eds): Handbook of Clinical Neurology. Vol 18. New York, Elsevier, 1992, pp 479 526. DOOLING EC, RICHARDSON EP JR: Delayed encephalopathy after strangling. Arch Neurol 33:196, 1976. EAYRS JT: In uence of the thyroid on the central nervous system. Br Med Bull 16:122, 1960. FARRELL RJ, KELLY CP: Celiac sprue. N Engl J Med 346:180, 2002. FERRACCI F, MORETT OG, CANDEAGO RM, et al: Antithyroid antibodies in the CSF. Their role in the pathogenesis of Hashimoto s encephalopathy. Neurology 60:712, 2003.
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folder from the first installation. Delete these files before reinstalling Exchange. For more information, see KB article Q256618.
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